Get Helpful Information About The Treatment Of The Inflammation Of The Trigeminal Nerve Inside This Post.

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Get Helpful Information About The Treatment Of The Inflammation Of The Trigeminal Nerve Inside This Post.

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The inflammation of the trigeminal nerve is one of the most disabling and painful conditions. It combines several types of pain with different mechanisms and, consequently, optimal approaches to treatment:
• Trigeminal neuralgia of type 1: there is more penetrating and recurrent pain.
• Trigeminal neuralgia of type 2: it is less common, the pain is constant, burning, has the boring character.
• Neuropathic pain of the trigeminal nerve: it is related to direct injury of the trigeminal nerve and / or pathways of the trigeminal nerve.
• De-afferent pain of trigeminal nerve: a pain in the face area, despite the lack of sensitivity in it – there is a painful anesthesia (anesthesia dolorosa).
• Symptomatic trigeminal neuralgia: the disease that is often connected with multiple sclerosis.
• Postherpetic trigeminal neuralgia: the pain caused by the outbreak of the herpes zoster virus (herpes zoster), usually it occurs in the area of V1 branch of the trigeminal nerve and, more often older people suffer from it.
• Neuralgia of the crankshaft (geniculate) nerve: it is paroxysmal and shooting pain in the pigeon’s ear.
• Neuralgia of the glossopharyngeal nerve: the pain occurs in the throat and tonsils that is launched with swallowing.

The causes of breakdowns in the trigeminal nerve system are diverse. For instance, virus, mechanical compression of nerve tissue by altered vessels, tumors, inflammatory processes, trauma etc. The cause can not be determined in some cases,.

Speaking about the mechanisms, we should know that violating of the myelin sheath integrity of nerve fibers with the violation of the sensitive information accuracy transmitting almost always happens. Information from the fibers that transmit a sense of touch, heat, taste, etc., jumps to the fibers that are responsible for pain signals (P-type fiber). It is an “alarm” signal for the central structures of pain perception. But there is no fire. There is shorted in the “fire alarm”.

Speaking about the diagnosis we must conduct clinical and neurological examination. The general and biochemical blood tests are required for patients receiving long-term carbamazepine. Magnetic resonance imaging of the brain can detect vascular, neoplastic, inflammatory and cicatricial changes that dictate different therapeutic approachs.

We can use the drug treatment. Recently, at the disposal of physicians low-toxic and effective anticonvulsants came: Topamax, Neurontin, Neiralgin, Lyrica.

Baclofen is rarely used to treat trigeminal neuralgia. Application of fenibute, pantogam, sodium hydroxybutyrate, mieglinola glycine has a weak evidence base.

Amitriptyline (Elavil) is an excellent product from a group of tricyclic antidepressants, such as desipramine (Norpramin), imipramine (Tofranil) and nortriptyline (Aventyl, Pamelor), but has many contraindications and monaminoksidazy inhibitors (MAOIs) are most often used then: isocarboxazid (Marplan), phenelzine (Nardil), trantsipromin (Parnate), or selective serotonin reuptake inhibitors (SSRIs): citalopram (Celexa), escitalopram (Lexapro), fluoxetine (Prozac), paroxetine (Paxil, Pexeva), sertalin (Zoloft).

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